ALUCINOSIS ORGANICA PDF

Psicopatología | Alucinosis |. More information Alucinosis | Síndrome de Charles Bonnet o alucinosis orgánica: a propósito de un caso. Download Citation on ResearchGate | Organic alucinosis or complex visual hallucinations. Charles Bonnet Syndrome: Conceptual revision and review of a case. Resumen El síndrome de Charles Bonnet, o alucinosis orgánica, fue descrito por primera vez por Charles Bonnet en Se trata de una entidad clínica en la.

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Muchos de estos efectos se producen incluso en dosis muy bajas, por lo que actualmente no hay una dosis de consumo que sea segura o que produzca un efecto beneficioso neto sobre la salud a nivel poblacional3. Parte del alcohol se metaboliza en estomago por la alcohol-deshidrogenasa ADH que es menor en la mujer. Atraviesa con facilidad todas las barreras. A veces se precede de crisis convulsivas generalizadas de abstinencia. Se altera la memoria declarativa memoria directamente accesible al conocimiento pero no la memoria de procedimiento habilidades aprendidas, no declarables.

Debe realizarse el diagnostico diferencial con otros tipos de demencia. Se acepta que 1 UBE equivale a 10 gramos de alcohol. Consumos mayores de 60 gr. Valores mayores de 96 son considerados indicativos de abuso de alcohol. El VCM disminuye lentamente con la abstinencia y se normaliza en meses, volviendo a elevarse tras la ingesta Se eleva con consumos mayores de gr.

La especificidad de la CDT es mayor que la de los otros marcadores. Su utilidad para mantener la abstinencia es menor Abstract We describe the fundamental aspects of the diagnosis and treatment of alcohol dependence. We analyse the steps taken for a systematic diagnosis DSM-IV and ICD criteria, diagnostic questionnaires on alcohol dependence and biological markers and the treatment for alcohol cessation. Alcohol, dependence, diagnosis, treatment.

Throughout history, alcohol consumption has been considered to be beneficial, especially in our country where cultures and traditions revolve around wine. During the last few decades, developments have been made into the study of the damage caused by alcohol consumption.

The relation between alcohol consumption and its impact depends on the quantity of alcohol intake and the pattern of consumption. The bio-chemical effects, depending on the amount of alcohol intake and the pattern of consumption, are closely linked with chronic effects at an individual level, whereas intoxication is associated with acute effects such as accidents, violence and social conflict.

Alcohol dependence resulting from long-term consumption has severe consequences, both acute and chronic, and is linked to more than 60 physical and mental illnesses, as well as numerous social problems affecting those around people who drink, with violence being one of the clear repercussions.

Aetiology and pathogenesis of alcohol-related disorders The factors involved in the aetiology of alcoholism are genetic, psycho-social and environmental. The phenomena which contribute to the development of dependence are those directly linked to the reward system positive reinforcement and those which involve other systems whereby alcohol consumption acts as a negative reinforcement.

Both processes involve different transmission systems. In alcohol dependence, this involves the anatomical reward circuits; the medial forebrain bundle, the ventral segmental area, the lateral hypothalamus and areas of the prefrontal cortex. Also involved are the neurotransmitters dopamine, noradrenalin, serotonin, glutamate, endogenous opioids, GABA and the calcium channels.

Acute alcohol orrganica causes sedation and euphoria by releasing dopamine, inhibiting the calcium orgamica, inhibiting the NMDA glutamate receptors N-methyl-D-aspartateand potentiating GABA.

EXAMEN MENTAL

During withdrawal, a state of neuronal excitement occurs, which involves an increase in the number of NMDA receptors and calcium channels, and a decrease in GABAergics. NMDA receptor activity in the hippocampus may cause convulsions and an increase in dopamine release in the hippocampus may cause hallucinations and delirium4. Its molecule is small and non-polar, which allows it to pass easily through biological membranes.

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One gram of alcohol supplies 7. The rate of absorption depends on the presence of food in the stomach, and the quantity and type of alcohol ingested. Part of the alcohol is metabolized in the stomach by the alcohol dehydrogenase ADHwhich alucknosis lower in women. It is rapidly distributed throughout the body, except for the fatty tissue, and reaches its maximum concentration level allucinosis minutes after ingestion.

It easily crosses all barriers, is predominantly hepatically metabolised and is mainly eliminated renally. Alcohol produces considerable pharmacological effects depending on blood alcohol organicca, type of consumption acute or chronicand the characteristics of the individual.

The duration of an episode of intoxication depends on factors such as the quantity and type of alcoholic beverage consumed the speed of ingestion, and absorption depending on whether it ogranica consumed with or without food. When intoxication becomes progressively more intense, it goes through aulcinosis stages; psychomotor excitation, in-coordination with mental depression, and coma. It may also cause amnesia in relation to the events occurring during the period of intoxication.

As tolerance develops, the motor, sedative and anxiolytic effects of alcohol decrease in intensity5.

Table 1 Table I: Actions of alcohol – Blood alcohol concentration 0. Euphoria Excitement Talkativeness Decreased intellectual capacity Slower reaction times – Blood alcohol concentration 0.

Emotional excitability Decreased visual acuteness Decreased judgement capacity Relaxation Sense of well-being Impaired eye movement – Blood alcohol concentration 0.

Slower general reactions Impaired reflexes Motor impairment Euphoria, relaxation, sense of well-being Impulsiveness Overestimation of own capabilities – Blood alcohol concentration 0. Severe drunkenness Severely impaired reflexes with delayed response Loss of control and problems with coordination Difficulty focusing Decreased alertness and perception of danger – Blood alcohol concentration 1. Alcohol dependence syndrome DSM-IV A maladaptive pattern of alcohol consumption leading to clinically significant impairment or distress, as manifested by three or more of the following criteria occurring at any time during the same month period: Alcohol is frequently consumed in larger quantities, or over a longer period than initially intended.

Persistent desire, and unsuccessful attempts to control or stop drinking 5. Much time is spent obtaining, consuming and recovering from the effects of alcohol zlucinosis. Neglect of other pursuits because of time spent drinking 7.

Persisting with drinking behaviour despite clear evidence of harm Table III: Recent alcohol intake B. Clinically significant maladaptive psychological changes in behaviour that occur during intoxication, or within minutes of ingesting alcohol C.

At least one of the following signs: The symptoms are not caused by medical illness, nor are they caused by the presence of another mental disorder Table IV: Cessation or reduction of prolonged alcohol consumption in large quantities B.

Development of two or more of the following symptoms in the hours or days after achieving criteria A: Hyperactivity perspiration or pulse rate of over Severe trembling of the hands Nausea or vomiting Anxiety Psychomotor agitation Visual, tactile or transitory auditory hallucinations, or illusions Grand mal seizure epileptic seizure C.

These symptoms cause clinically significant distress or impairment of work-related activity D. Harmful consumption ICD refers to the existence of medical or psychological problems resulting from alcohol consumption, irrespective of the quantity consumed.

Alcohol dependence syndrome refers to the existence of behavioural and physiological symptoms which indicate a loss of control over consumption and a tendency to continue drinking despite the adverse consequences. The pathological link with alcohol develops over years into an expression of physical and psychological changes, resulting from long-term intoxication.

At this time, the changes caused by alcohol are sub-clinical; the psychiatric changes appear later. Traffic accidents are the main problem caused by alcohol intoxication. Others include criminal acts, suicide, accidents and falls resulting in fractures, and traumatic brain injuries.

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Exposure to the elements can cause freezing or burns. Suppression of the immunological mechanisms may act as a predisposition to infection.

Pathological intoxication is defined by significant behavioural changes, which in most cases includes aggression appearing almost immediately after consuming quantities of alcohol that would be insufficient to cause intoxication in most individuals. The behaviour is untypical in the sense that it does not occur when the individual has not consumed alcohol.

Alcohol withdrawal delirium delirium tremens is characterised by disturbance of consciousness, agitation, severe trembling of the hands, frequent micropsic and animal hallucinations both spontaneous and inducedoccupational delirium and autonomic hyperactivity. It is sometimes preceded by generalised convulsive withdrawal seizures. It usually begins on the second or third day after alcohol intake has ceased or has been reduced, although it may occasionally occur beforehand, or after a week of withdrawal.

The syndrome usually develops in days, unless any other problems arise due to an association with other illnesses. The first episode of this change usually occurs between 5 and 15 years after the establishment of a persistent habit of drinking alcohol. Having a concomitant physical illness may act as a predisposition to this syndrome.

Alcoholic hallucinosis is an organic hallucinosis that develops and resolves rapidly within around 48 hours and causes vivid and persistent hallucinations audio and visual after alcohol withdrawal or reduction in an individual who appears to be dependent on alcohol but does not experience delirium. The first episode of this change usually occurs after 10 years of excessive alcohol intake.

The disorder may last for several weeks or months. This syndrome is characterised by anterograde and retrograde amnesia, confabulation, changes in attention and concentration, lack of insight into the condition, apathy and indifference.

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There are changes to the declarative memory the memory that stores factual knowledge but not the procedural memory acquired skills that can be automatically recalled.

There are also changes to the episodic memory aluconosis specific events but not the semantic memory language, rules, relationships and general principles. The existence of alcoholic dementia is controversial. Possible aetiology would be the extra vulnerability resulting from thiamine deficiency and the direct neurotoxin effects of alcohol.

alucinosis de escena retrospectiva – English Translation – Word Magic Spanish-English Dictionary

The characteristic symptoms of aphasia, paraxial and agonise are only observed in one third of cases. The presence of ataxia and peripheral neuropathy is very common. Typical deficiencies are complex reasoning, planning, abstract thinking, judgement, attention and memory.

The condition should be diagnosed differently from other types of dementia. Although the deficiencies may remain, they can somewhat improve if sobriety is maintained for several years.

Foetal Alcohol Syndrome FAS is an embryo footpath that presents in the children of alcoholic mothers. It is characterised by slow pre and post-natal growth, mental and psychomotor deficiency, facial dysmorphia with microcephaly, microphthalmia or small palpebral fissures, thin upper eyelids, and flattening of the maxillary region. Other malformations include haemangioma, slight abnormalities of the joints and extremities, genital abnormalities, heart defects and single palmer creases5, 8, 9.

Diagnosis of alcoholism The diagnosis of alcoholism is made using a correct anamnesis, along with a correct clinical examination. It is necessary to assess the quantity and frequency of alcohol intake, and diagnostic questionnaires can be useful for this. A thorough analysis will be conducted using biological markers and an appropriate dual diagnosis of previous or associated psychopathologies.